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    An examination of the molecular mechanisms controlling the tissue accumulation of conjugated linoleic acid (CLA) in cattle

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    Author
    Waters, Sinead M.
    Hynes, A.C.
    Killeen, Aideen P.
    Moloney, Aidan P
    Kenny, David A.
    Keyword
    Cattle
    Conjugated Linoleic Acid (CLA)
    Gene expression
    mRNA expression
    Cell line resource
    Date
    01/12/2008
    
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    URI
    http://hdl.handle.net/11019/859
    Citation
    S.M. Waters, A.C. Hynes, A.P. Killeen, A.P. Moloney, D.A. Kenny, An examination of the molecular mechanisms controlling the tissue accumulation of conjugated linoleic acid (CLA) in cattle, End of Project Report, Teagasc, 2008.
    Abstract
    Long chain n-3 polyunsaturated fatty acids (n-3 PUFA) and conjugated linoleic acid (CLA) have demonstrable and potential human health benefits in terms of preventing cancer, diabetes, chronic inflammation, obesity and coronary heart disease. Supplementation of cattle diets with a blend of oils rich in n-3 PUFA and linoleic acid have a synergistic effect on the accumulation of ruminal and tissue concentrations of trans vaccenic acid (TVA), the main substrate for ?-9 desaturase which is responsible for de novo tissue synthesis of the cis 9, trans 11 isomer of CLA. This dietary strategy translates into increases in milk concentrations of CLA in dairy cows; however, concentrations in the muscle of beef animals have not always been increased. There is an apparent paradox in that n-3 PUFA supplementation enhances ruminal synthesis of trans-vaccenic acid (TVA), but then inhibits its conversion to CLA possibly through altering the activity of ?-9 desaturase. Recently, the promoter regions of the bovine ?- 9 desaturase gene has been isolated and analysed and has been shown to contain a conserved PUFA response region.
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    Food Chemistry & Technology
    Food Programme End of Project Reports
    AGRIP End of Project Reports
    Animal & Bioscience
    Animal & Bioscience

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